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Even the behaviors of the treated mice were different.
Interestingly, the TREome landscapes of C3H/HeJ (toll-like receptor-4 [TLR4] mutant) inbred mice were different from its control C3H/HeOuJ (TLR4 wild-type) strain.
The spine changes observed in the cerebella of the EphB-deficient mice were different from those seen in the hippocampus of EphB1−/−, EphB2−/−, and EphB3−/− triple knockout mice and various combinations of EphB−/− double knockout mice, where decreased spine density and abnormal spine morphologies were observed [7].
Microarray analyses revealed that, 10 days post-infection with S. japonicum, the gene expression profiles in the lungs of the non-susceptible hosts, M. fortis and rats, and in the susceptible host, mice, were different from the uninfected animals; this was consistent with our previous microarray results between uninfected M.fortis and mice and those challenged with S. japonicum for 7 d [18].
Taken together, none of the MG abnormalities observed in MMTV-cortactin mice were different from WT FVB/N mice.
However, the background strains of the mice were different between our study and theirs (ICR vs. CD2F1, respectively).
Similar(44)
As expected the body weight of male and female mice was different and there was a minor but statistically significant difference in baseline albuminuria (Figure 1A).
At 72 hours, the increase in inflammatory cells seen histologically in the airways of Nlrc4−/− mice is different from the bronchoalveolar lavage data that has no difference.
The percentage of albuminuric mice was different in the individual transgenic lines ranging from 23 45%.
The pattern of induction of NQO1 in the SCD mice was different from that observed for HO-1.
In conclusion, the present study has demonstrated that different DC subsets from BCG-infected mice are different in capacity to generate protective immunity.
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