Sentence examples for mice we would from inspiring English sources

For Gsntm1Djk/Gsntm1Djk Whrnwi/+ mice we would predict a phenotype that corresponds to Gsntm1Djk/Gsntm1Djk mice, and again this is observed (Figure 5b).

Therefore, based on findings in our Ghsr KO mice we would favour a purely direct action on peripheral tissues by peripherally administered UAG.

If there were some FD present in MASP-1/3 –/– mice we would expect to be able to demonstrate some AP activity.

Because the Venus transgene was driven by the gcg promoter in these mice, we would have expected close to 100% overlap in these graphs.

Had the levels of incretins been increased in the knockout mice, we would have expected certain metabolic consequences, such as reduced body weight and improved glucose tolerance (Suzuki et al., 2012).

It is impossible to exclude the possibility of trans effects from these loci, however since they were present but not fixed in both test and control mice we would have had very low power to detect trans effects from the non-target loci, therefore we have disregarded possible effects from these loci in the subsequent discussion.

I'm sure that if we'd been able to get the word out to thousands of people with the click of a mouse, we wouldn't have hung up our placards and called it a day; that drudge work absorbed the lion's share of our time and our capacity to think up new and exciting ways to make change.

If the structure of transcriptional regulation is similar between human and mouse, we would expect a similar, limited number of TSRs to be needed to summarize the same amount of variance in total expression as seen in the PCA performed on human and mouse expression data separately.

"The joke is if we were trying to cure mouse cancer we would have done it 30 years ago," said Dr. Donald Bergstrom, a vice president at Sanofi.

If GM is superior in NMR (and human) compared with mouse, then we would expect to find that reflected by their germline mutation rates.

In line with a worsening of glucose tolerance in our mouse models, we would predict that a beta cell-specific Tcf7l2 loss of function would lead to the same phenotypes described by Rutter and colleagues.