Sentence examples for mice we stimulated from inspiring English sources

To examine the synergism between our TLR9 and STING agonists for early (innate) IFN-γ induction in mice, we stimulated mPBMCs in vitro with K3 CpG, cGAMP, or the combination.

To determine whether Th-17 cell maturation and/or IL-17 secretion was affected in Df11(1)/+ and Dp11(1)/+ mice, we stimulated naïve splenic T cells (Th0 cells), in culture with IL-6 and TGF-β to become Th-17 cells and used flow cytometry coupled to intracellular immunostaining and real-time PCR to determine production of IL-17.

To mimic exercise in the mice, we tetanically stimulated the tibialis anterior and extensor digitorum longus muscles of mice that have been previously used for the above in vivo physiology recordings and measured AMPK activation levels.

In order to test if the circulating levels of CTLA4IgG were sufficient to have an effect on other secretory epithelia such as the loss of lachrymal gland function in the C57BL/6.NOD- Aec1Aec2 mice, we measured stimulated tear flow at the end of the study.

To further establish the role of TLR2 in modulating the progression of atherosclerosis, we stimulated mice with the TLR2 agonist known as FSL-1.

To study the differential role of QC and isoQC in vivo, we stimulated mice from both knockout lines by a peripheral (intraperitoneal; i.p).

To investigate the ability of the human CD8+ T cells reconstituted in the hNOK mice to produce these cytokines, we stimulated splenocytes isolated from these mice with PMA and ionomycin in vitro and measured the production of the above cytokines.

To determine whether iTregs also express surface LAP, we stimulated mouse CD4+CD25− T cells in the presence or absence of recombinant TGF-β and checked for surface LAP expression.

To explore whether PP1cγ participated in thrombin signaling downstream of mouse thrombin receptor PAR4, we stimulated platelets with protease-activated receptor 4 activating peptide (PAR-4AP) and assessed fibrinogen binding.

We stimulated mouse coronary vascular endothelial cells with PGN or LPS for 24 h and examined cellular ICAM-1 protein levels.

To test this hypothesis, we stimulated normal mouse mammary epithelial (NMuMG) cells with TGF-β1 and monitored changes in actin stress fiber formation by immunofluorescence.