Sentence examples for mice we show from inspiring English sources

By engineering TGF-β and BMP-reporter mice, we show that TGF-β2 signaling antagonizes BMP signaling in HFSCs but not through competition for limiting Smad4-coactivator.

Using genetically engineered mice, we show that precocious Norrin production leads to premature retinal vascular invasion and delayed Norrin production leads to characteristic defects in intraretinal vascular architecture.

Using an orthotopic pancreatic cancer model in syngeneic C57BL/6 mice we show that the local administration of HCA-EF-ZP achieves better tumor/liver ratio of luciferase production than the intravenous route.

By using genetically engineered mice, we show that the APC/C cofactor Cdc20 is essential for anaphase onset in vivo in embryonic or adult cells, including progenitor/stem cells.

In a model system using immunodepressed mice we show preference of a mouse embryonic mesenchymal cell line (C3H/10T1/2) for specific implantation sites and biomaterials during a prolonged in vivo growth period (3 months).

By using donor skin from these LC-deficient mice, we show that LCs are not required for rejection of major (FVB → B6) or minor (H-Y, male → female on B6 background) antigen-mismatched skin grafts.

Using FVB mice we showed, that daily single injections of quinine (10 mg/kg, i.p).

Here, using a modeling approach in humanized mice, we showed that human lymphoid development stemmed from distinct populations of CD127− and CD127+ early lymphoid progenitors (ELPs).

Using human blood, serum, and urine spiked with WNV and mouse blood and brain tissues from Karshi virus-infected mice, we showed that these clinical matrices did not inhibit the detection of these viruses.

Using global microRNA expression profiling in hippocampus of humanized BDNF Val66Met knock-in mice we showed that this variant results in dysregulation of at least one microRNA, which in turn affects downstream target genes.

By using Irf8+/– mice, we showed that the reduction was dependent on levels of the Irf8 gene.