Sentence examples for mice we postulated from inspiring English sources

Sle2c2 mice, we postulated that huG-CSF treatment should eliminate the difference in cGVHD response between B6 and B6.

Because the expression of UNC5B was localized to the sprouting vessels and was significantly elevated during the active angiogenic period of the OIR mice, we postulated a role of UNC5B in hypoxia-driven neovascularization.

Since NK cells were the only cell type shown to be critical for tumour rejection in both B6 and RAG /– mice, we postulated that Treg cells inhibit the activity of B16FasL-induced NK cells.

Given the histological and functional similarities of the mammary epithelium as well as the requirement of identical molecular pathways for the development of mammary glands in humans and mice, we postulated that the human genome might also contain an orthologous promoter that contributes to the transcriptional regulation of the AKT1 gene.

Due to the clear phenotype of the cIAP-1 knockout mice, we postulate a non-redundant role of cIAP-1 in chlamydial lung infection.

Because PyMT-driven tumors grow so much more rapidly than those in Neu mice, we postulate that tumors from PyMT mice are highly dependent on glycolysis for ATP production and to generate building blocks for cell division.

In view of the relatively high predictive performance of antibody binding profiles of serum samples from healthy BALB/c mice, we postulate that these sera exhibit properties of unbiased antibody mixtures.

Because of our previous findings concerning neutrophil extravasation in DHEA treated mice, we postulate that DHEA has either a direct or an indirect effect on the expression of adhesion molecules on leukocytes or endothelial cells.

When we consider the overall gene expression profile of tumors from Neu/S14 mice, we postulate that overexpression of S14 is associated with reduced levels of genes associated with early mammary stem cells and with breast cancer invasion and metastasis, as well as the elevated expression of genes involved with alveolar epithelial differentiation.

Based on our studies showing an artificial partial inactivation of FGF8 signaling in the young and adult mouse, we postulate that FGF8 signaling may similarly be involved and individuals with inactivating mutations in Fgf8 may be predisposed to affective disorders.

Because endogenous androgens are derived solely from the ovaries in female mice (20), we postulated that the observed difference in atherosclerosis between ARKO and control mice fed an HFD would be abolished in ovariectomized mice.