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Through lentiviral rescue in hotfoot mice, we noted a recovery of PC-PF contacts in the distal dendritic domain.
In the tumors of cyclopamine-treated mice, we noted a marked reduction in murine Ang-1, IGF-1, and PDGF-B mRNA, factors known to stabilize the neovasculature.
Interestingly, in the rH4/Ad-H4 vaccinated mice, we noted important differences between the CD4 and CD8 T cell recall responses.
In our breeding colony of SOD-1 (G93A) transgenic mice we noted absence of typical ALS-like symptoms in a female mouse at a time point where mice from the original SOD-1 (G93A) line usually suffer from strong gait impairment and reduced motor function.
Compared with the vehicle-treated mice, we noted significantly inhibited recovery in the muscimol-treated mice.
During handling of Bace1−/− mutant mice, we noted that their movement was altered.
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For the Mup1/2 primer set, compared with SC WAT and RP WAT of ob/ob mice, we note an average 60-fold (p < 0.001) higher transcripts expression for wild type SC WAT and an average 230-fold (p < 0.001) higher level of transcripts for wild type RP WAT (p < 0.001).
Similar to the mouse, we noted a second population of delaminating cells in the chicken neural ectoderm.
Consistent with the results of Schlosser et al. (2008) in Xenopus and Zou et al. (2004, 2008) in mouse we noted that loss of eya1 mRNA alters the expression of other otic genes.
Human BLyS is required to initiate engraftment of B cells from human PBL in NOD rag1−/−Ppf−/− mice and we noted that mice receiving BLyS for only 7 days had lower amounts of B cells compared with mice receiving 14 days of treatment (20 25% vs 30 60%, determined by comparison of overall CD20 staining of histology sections, Table 2).
The in vitro observations of increased autophagy lysosome pathway activity during cancer cachexia correlated with skeletal muscles isolated from C26 tumour-bearing mice, as we noted elevated conversion of LC3-I into LC3-II in cachectic mice when compared with the controls.
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