Sentence examples for mice we focused from inspiring English sources

To provide the first quantitative analysis of axonal reorganization after focal injury in the brain of living mice we focused on the dynamics of surviving L6, L2/3/5 and TCA segments.

To validate the RNA-seq results functionally and to initiate signal transduction analyses of Nampt mediated pathways in CIA Nampt+/− mice, we focused on GM26870, a differentially expressed long non-coding RNA (lncRNA), to examine whether it may be a component underlying the protective role of Nampt knockdown in arthritis.

Since, as reported by these groups, mutation of these domains individually does not affect the interaction of BCR-ABL with its substrates, nor its ability to induce leukemia in mice, we focused our investigation on the triple mutant encompassing each of these component mutants in our studies.

Searching for factors that could play a role in TB progression in F2 mice, we focused our attention on our previous data that detected differences between parental I/St and A/Sn mice with respect to the intensity of lung tissue inflammation.

Since no motor phenotype was detected in Surf1 −/− mice, we focused our analysis on the other two COX-defective models that did show motor impairment.

To elucidate the various signaling molecules and growth factors involved in the anti-angiogenic effect of SMT in the OIR mice, we focused on HIF pathway-mediated expression.

Although forebrain weight data are now available for both the original "Taylor" BXD mice and the "Williams" BXD mice, we focus only on the "Taylor" BXD mice.

As most experimental work in topographic map formation is now undertaken in mouse, we focused on curating the experimental data available in the literature including in wild type, Isl2-EphA3ki/ki, Isl2-EphA3ki/+, triple ephrin-A2,A3,A5 knock-out, and Math5−/−.

In order to track for the neonatal and embryonic paralleles in the adults of the MRL/MpJ mouse, we focused on the transcripts which were differentially regulated in newborns, embryos and the MRL/MpJ adults relative to the adults of the control strains.

Finally, to remove residual bias from the mouse strain, we focused on mice with SJL (39 founders) and (C3H/HexSJL F1 (four founders) background as we had a strain specific reference genomes for these stains before ENU mutations were introduced.

Several genes were significantly upregulated in these mice, and we focused on the S100a9 gene, whose expression was markedly increased.