Sentence examples for mice we characterized from inspiring English sources

After 7 9 weeks of propagation, prior to transplantation into the brains of EAE mice, we characterized the phenotype and in vitro differentiation potential of the cells within the spheres.

Using lymphocytic choriomeningitis virus (LCMV) peptide gp33-specific CD8+ T cells derived from T cell receptor transgenic mice, we characterized the metabolic phenotype of proliferating T cells that were activated and expanded in vitro in the presence or absence of rapamycin, and determined the capability of these rapamycin-treated T cells to generate long-lived memory cells in vivo.

With intrathecally (IT) delivered BoNT-B in C57B/l6 mice, we characterized the effects of such block on the release of substance P (SP) from spinal afferent nociceptors (as measured by neurokinin-1 receptor, NK1-R, internalization), spinal neuronal activation (as indicated by spinal C-Fos expression) and nociceptive behavior after intraplantar (IPLT) formalin.

Using an excisional wound model in mice, we characterize the appearance, growth, and maturation of blood vessels in an Integra™ graft over 28 days after surgery.

In addition, using antibiotic intervention and germ-free mice, we characterize the effect of the microbiome on the ILC regulatory landscape and determine the response of ILCs to microbial colonization at the single-cell level.

Using the IFNγ-YFP mouse we characterized the level of YFP expression in various cell populations following immunization with gp120.

Using the Lewis lung carcinoma (LL2) implantation mouse model, we characterized the hematopoietic compartment in the tumor stroma.

Using different genetic mouse strains, we characterized their kinetics of recruitment from the bone marrow to the lungs and show that this recruitment is independent from a CCR2-mediated signal.

In order to select time-points for transcriptional analysis that are matching the onset of neuropathy in Ins2Akita/+ mice, we first characterized the development of DPN in this model.

Since we did not observe choroid plexus hyperplasia in NHERF1-deficient mice, we further characterized the ependymal cells.

Prior to performing the ex vivo experiments with cells isolated from AD-induced mice, we firstly characterized CD4+ T cells and CD19+ B cells isolated from AD-induced mice by comparing with cells isolated from normal mice.