Sentence examples for mice we assessed from inspiring English sources

On a set of laboratory mice, we assessed how wear related to age and food consistency affected molar geometry and topography.

Here, monitoring T-cells restricted by HLA-A*02 01 HLA-A*02 01in mice, we assessed whetransgenict the tHIVconsv design (HIVconsv with a tissue plasmicegen activator leader sequence) benefits from combining weth assessedmenting conserved mosaic immunogen tHIVcmo, and compared the bivalent immunization to that whetherivalent corserved mosaic vaccinot.

Since AchR clustering is efficiently maintained in MCK-UCP1 mice, we assessed whether axonal regeneration upon injury was normal.

To begin to elucidate the mechanisms responsible for impaired glucose tolerance in caCnRIP mice, we assessed insulin secretion in vivo and in vitro.

To further characterize the megakaryocytic compartment in SHIP deficient mice, we assessed the presence of early MKP, immuno-stained as Lin−c-Kit+CD41+, and of more differentiated MK, identified as Lin−c-Kit Lin−c-Kitigure 2A) [34].

Furthermore, we detected NF-κB binding in colonic nuclear extracts from all but one of the CONV-R mice we assessed, but never in any extract from a GF mouse.

By directly comparing the relative expression ratio of orthologous genes between human and mouse, we assessed how closely the mouse models mimic the gene expression activity of two human HCC subclasses.

To establish whether human oocytes demonstrate the same molecular changes as the mouse, we assessed expression of these targets in GV oocytes obtained from patients undergoing IVF treatment.

In view of previous data suggesting neuronal dysfunction rather than neurodegeneration in our A53T-SNCA transgenic mice [13], we assessed the effect of A53T-SNCA overexpression on dopaminergic neurotransmission by measuring DA steady state levels in striatal tissue homogenates using high pressure liquid chromatography (HPLC).

As DX5− NK cells are reported to include CD11b− immature NK cells and NK1.1 is not expressed in BALB/c strain mice 25, we assessed only the CD11b+ mature phenotype of NK cells.

Because activation of the AMPK pathway provides an alternative stimulus for increased glucose disposal and AMPK phosphorylation and expression were reduced in myostatin null mice (58), we assessed activating phosphorylation of AMPK at T172 and phosphorylation of its substrate acetyl-CoA carboxylase (ACC) at the AMPK target residue S79.