The subcutaneous route of challenging mice was shown to be better than intraperitoneal injections.
The total effect of mice was shown to be positive for all of the cohorts.
A decrease in the bone formation rate in F508del Cftrtmicer mice was shown compared with control mice, independently of age and sex.
Cohabitation for 14 days with Ehrlich tumor-bearing mice was shown to increase locomotor activity, to decrease hypothalamic noradrenaline (NA) levels, to increase NA turnover and to decrease innate immune responses and decrease the animals' resistance to tumor growth.
Cohabitation for 14 days with an Ehrlich tumor-bearing mice was shown, among others, to increase locomotor activity, and hypothalamic noradrenaline levels and turnover, to decrease the innate immune responses and animal resistance to tumor growth.
Overexpression of Hdac6 in the testis of Hdac6 transgenic mice was shown by immunohistochemistry, immunofluorescence microscopy and qPCR analysis (Fig. S2E, S2F and S2G).
Down-regulation of SSTR2 after a single injection of octreotide in AR42J-bearing CB-17 SCID mice was shown already after 0.5 h and with a complete cell surface recovery of SSTR2 after 16 24 h [37].
Conditional deletion of Sav1 or Lats1/2 in neonatal mice was shown to promote heart repair and extend the regenerative potential following cardiac resection at P8. Furthermore, Hippo deficiency led to increased cardiomyocyte proliferation and myocardial protection in response to MI either at P8 or in 8-week-old adult mice.
The codominantLps d allele of C3H/HeJ mice was shown to correspond to a missense mutation in the third exon of the Toll-like receptor-4 gene (Tlr4), predicted to replace proline with histidine at position 712 of the polypeptide chain.
In the intervening years, a virus in mice was shown to be related to the virus causing scours.
Flagellin-induced protection against radiation, chemicals and pathogens in mice was shown to be TLR5 and MyD88-dependent [33].
