miR-223 expression in miR-223−/+ mice was lower than that in WT mice and had no obvious effect on MTDs.
(b) The ratio of liver weight to body weight of the hMSG-EpiPCs treatment mice was lower than that of the PBS-treated mice.
Intensity of staining with UEA-I in secondarily infected mice was lower than in rIL25-treated mice which might be related to lesser quantity of IL-25 in the environment.
This result in the SCGx group was confirmed by a flow cytometric analysis of cells harvested 18 h after corneal abrasion (Fig. 3f, left and middle); the number of Ly6G+ neutrophils in the wounds of SCGx-treated mice was lower than in the control mice (Fig. 3f, right; factorial-design ANOVA, P < 0.01).
The results show that the concentration of GM-CSF in the lungs of RNF13-KO mice was lower than that in WT mice after bearing B16F10 cells (Fig. 4D).
First, the basal level of the HPA axis activity in tg mice was lower, compared to control mice, even though the difference was not significant (Fig. 6).
Although not statistically significant, the tumor multiplicity of SCC I in K5-VILIP-1 mice was lower than in WT mice (0.68 SSCI/mouse versus 0.88 SCCI/mouse).
After 48h of fasting, the glycogen content in liver of wild-type mice was lower than taht of GLUT2-loop transgenic mice (Fig. 4A).
The percentage of VCAM-1 positive vessels in wt and ApoE−/− mice was lower in diabetic animals (p<0.05 and p<0.001, respectively), whereas no differences were observed in the TNFα deficient genotypes (Figure S1).
In agreement with the findings in cultured cells, the phosphorylation of p38MAPK in lysates from A02 mice was lower than that detected in B05 extracts but slightly increased when compared to lysates from nontransgenic controls (Figure 6A).
In addition, the expression level of DPPI mRNA of PLN CD8+T cells from CatL siRNA-treated mice was lower than that from control siRNA-treated mice (Figure 5E).
