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Moreover, the hyperparathyroidism observed in CKD mice was also corrected.
The survival rate of the infected mice was also registered.
What's more, astrocytosis in brains of APP/PS1 co-transgenic mice was also relieved.
Subtle thickening of proximal media/adventitia not typically seen in mice was also detected.
This hierarchy of CTL responses in mice was also reflected in an MHC stabilisation analysis.
Moreover, level of lipid peroxidation in ethanol with AXT-fed mice was also decreased compared with ethanol-fed mice.
An Escherichia coli based infection model in aging mice was also designed to validate the protective attributes of LF.
The pattern of protein expression in the prefrontal cortex of C57BL6N mice was also clearly distinct from 129X1/SvJ mice.
Lower atherosclerosis, as compared with control mice, was also seen if the gene expression of TAZ rather than YAP was decreased.
Our results showed that the expression of miR-200c-3p miR-200c-3p miR-200c-3p5N1-infecthe mice was alungmarkedly upregulatissueigure 5a).
Uptake in NSCLC xenografted mice was also observed.
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CEO of Professional Science Editing for Scientists @ prosciediting.com