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In contrast, PGI2, a prostanoid markedly increased at day 5 of pregnant wild type mice, was able to partially restore decidualization.
Consistent with these findings, the adoptive transfer of spleen cells from immunized mice to naïve syngeneic mice was able to transfer anti-tumor effect, suggesting that GP10025 33 peptide-specific immune response was induced.
Conversely specific overexpression of DMP1 in mice was able to resist mannitol-induced BBB reversible opening.
Moreover, Indoprocaf, but not sumatriptan, administered i.p. at sub-analgesic doses in mice, was able to abolish peripheral and central sensitization [7].
In both cases ischemia activated their expression and infusion of NC to ischemic mice was able to elicit a higher increase.
PrPSc purified from prion-infected mice was able to seed misfolding of PrPC in a protein misfolding cyclic amplification reaction, and mutant PrP aggregates from transgenic mice were toxic to cultured neurons.
Similar(34)
Mice that received MDPs and CDPs from wild-type mice were able to restrain PyMT-mCh-OVA tumor growth, similar to wild-type mice, whereas mice that received MDPs and CDPs from tumor-bearing mice were not able to restrain tumor growth (Fig. 3e).
Analysis was performed only if mice were able to walk at least 3 steps without stopping.
The quercetin mice were able to run as much as 37percentt longer than they had the week before.
By contrast, wild-type mice were able to recover after the toxic insult.
The Alzheimer's mice were able to recall the correct memory with a little help from the researchers.
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