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In control mice, the strength of the reaction was similar (P = 0.22, unpaired Student's t-test) to that obtained in five rats, as assessed from the gold particle densities in the main apical dendrites in the middle of the SR (8.6 ± 2.6 gold/μm, n = 3 control mice; Fig. 5A; Table 2), and showed a similar labelling pattern within the SR and SLM to that found in rats.
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Oh, I forgot to mention, the radiation gives each malevolent mouse the strength of fifty.
In mouse, the strength of NANOG binding (measured by the number of ChIP-PET ditags) was positively associated with the strength of POU5F1 binding to the same region according to our re-analysis of ChIP data [ 1] (Additional file 10).
Before the Morris water maze test, the weights of mice and the strength of mice muscle were measured to make sure there were no remarkable physical differences between Rib-injected groups and control ones (Supplementary Figures 4A and B).
Interestingly, however, there was a significant decrease in magazine entries in devalued BALB/cJ mice (although the strength of the LiCl-induced illness-food pairing seemed relatively weak in these mice and was not expressed on the long-term retention test).
In vivo experiments corroborate the estrogenic effects of enterolignans through preferably activating ERα in female mice, although the strength of the estrogenic effects varies in these studies (29– 31).
We conclude that the simplest explanation for the accelerated disease in mice generated from matings of Gurney WT and L126Z mice is that higher levels of WT SOD1 expression in Gurney WT mice simply increase the strength and frequency of interactions between WT and mutant proteins and thus increase the opportunities to produce toxic structures.
We have noted that the follicular type I B-cell population was preserved in Aiolos-deficient mice, in which the strength of BCR signaling was increased, but the number of splenic MZ B cells was markedly decreased, as well as MZ precursor B cells.
In another experiment with mice, the scientists documented the strength of the mouse antibody protection against SARS.
In one experiment, the scientists electrically stimulated nerve cells from the mouse brains, assessed the strength of synapses from Shank3 mutants and compared these to brain tissue from normal, wild-type mice.
Furthermore, a reduction in the rate of transfer from the A1 state to the A2 state in GluA1−/− mice may lead to actually enhancing trace conditioning, relative to control mice, due to increasing the strength of the A1 representation at the time at which the US is presented.
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