Sentence examples for mice suggests abnormal from inspiring English sources

The increased USV in maternal deletion mice suggests abnormal signaling behavior between mothers and pups that may reflect abnormal communication behaviors in human AS patients.

Similarly, in both adult APP/PS1 and old nonTg mice, a greater increase in LTP expression due to the addition of bicuculline compared to the lower increase in LTP in adult nonTg mice suggests abnormal enhancement of both excitation and inhibition.

Both genes are highly upregulated in p53 TSD /− mice, suggesting abnormal adipocyte composition in the skin of p53 TSD /− mice (Supplementary Figure S6).

The augmented late gestational enlargement of intraplacental arteries, an apparently blunted angiogenic response by capillaries, elevated labyrinth cell death rates (20), and reduced fetal weight gain relative to CD1 mice all suggest abnormal placental development in late gestation in B6 mice and highlights an important role for genetics.

Therefore, although LFPs in Tau mice were overall smaller, prominent theta oscillations were still present and qualitatively similar to those of WT mice, suggesting that abnormal theta oscillation is unlikely the cause of the difference between Tau and WT sequences.

No differences were identified in gross retinal anatomy (data not shown) or light-driven pupil constriction in Bdr mice suggesting that abnormal inputs from the eye are not the source of the circadian phenotype.

Overexpression of XBP-1 in B cells is sufficient to cause a monoclonal gammopathy of undetermined significance in mice, suggesting that abnormal expression of XBP-1 could be a predisposing factor for the development of myeloma (Carrasco et al, 2007).

Analysis of the temporal-expression profile of MBP (myelin basic protein) isoforms showed unexpected increases of the 14, 17 and 18.5 kDa isoforms in the sciatic nerve of 1-week-old Twitcher mice, suggesting an abnormal regulation of the myelination process during early postnatal life in this mutant.

Our findings of abnormal NMJ complexity in Cra1/+ mice suggests that dynein plays a role in development and maintenance of mammalian synapses as well.

Lack of abnormal phenotypes in these Muc16 knockout mice suggests that CA125/MUC16 is not required for normal development and reproduction.

Lack of any prominent abnormal phenotype in these Muc16 knockout mice suggests that CA125/MUC16 is not required for normal development or reproduction.