Soon headless mice started to appear.
Following 2 weeks of diet feeding, a cohort of mice started to receive the CYP2E1 inhibitor diallyl sulfide (100 mg/kg/d, i.p).
Three days later, a few mice started to walk in circles, indicating that neurons on one side of their brains were making dopamine.
"If scientists inhibited these pheromones in the child mice, adult male mice started to mate with these babies".
At the end of week 3, some mice started to undergo wasting.
6 10 days after infection, susceptible mice started to develop symptoms of cerebral malaria (CM).
However, when the SOD1G93A and SOD1G93A GarsC201R/+ mice started to show hindlimb weakness, these mice were weighed at least twice a week.
VDR−/− mice started to develop alopecia at 9 weeks of age, which later than some other VDR null strains [15], [20], [33].
Control mice started to recover by day 4 and all survived and regained a normal hematocrit by day 10 (Fig. 3B and C).
IRBC accumulation was assessed in isolated organs on days 7 and 8 post-infection (at the time when WT mice started to develop ECM).
However, at 24 h singly infected mice started to clear the bacterial infection in both the bronchoalveolar lavage (BAL) and lung-tissues, while co-infected mice failed to control bacterial proliferation at all sites tested (Fig. 2).
