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The crocidolite-exposed mice show higher number of mutations as compared to sham treated.
As observed in DM1 patients [4], dmpk−/− mice show higher concentrations of plasma insulin than wild-type mice in the glucose tolerance tests.
Clock mutant mice show higher number of mid gestation fetal reabsorption [28] and lower number of pups born when mice are kept under continuous darkness [29].
Since female mice show higher baseline hepcidin expression than males ([41]; Figure 9C), we used exclusively female littermates for the experiments.
A bias inherent to the doubleridge model is that these mice show higher basal Dkk-1 protein levels in brain tissue [46], although the Dkk1 gene is not fully responsive to inductive cues.
It has been proposed that leptin-deficient mice show higher food efficiency rates at least in part due to an impaired capacity of BAT to produce heat [2], [41].
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TXNIP knock-out mice show high levels of accumulated inactive PTEN with higher levels of activated AKT resulting in more glycolytic oxidation in their skeletal muscle [94].
GFP+ Schwann cells purified from GFP mice show high susceptibility to ML infection.
Sera from normal MA mice show high levels of a natural antibody capable of neutralizing the virus.
Kidneys of nephritic NZM2328 mice show high levels of expression of Th1-type cytokines like IFN-γ and IL-12 [ 67].
Under light-dark conditions and ad libitum feeding, αMUPA mice show high amplitude, appropriately reset circadian rhythms in peripheral clock genes [ 194].
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