Sentence examples for mice show abnormal from inspiring English sources

ΔHS1 mice show abnormal limp grasping indicating a neurological abnormality, ΔHS1 mice can develop severe hydrocephalus and exhibit retinal impairments.

Indeed, homeodomain transcription factors Dlx1 and Dlx2 knockout mice show abnormal differentiation of basal ganglia cells, and CT axons approach to the pial surface instead of a sudden turn to the basal ganglia as in control mice (Bulfone et al., 1993; Anderson et al., 1997; Qiu et al., 1997a; Garel et al., 2002).

Thus tangentially migrating neurons derived from the medial ganglionic eminence of Dcx KO or RNAi-treated mice show abnormal morphologies during migration in explant and slice cultures.

PACAP and PAC1 knock-out mice show abnormal behavior resembling psychiatric disorders [5] and some SNPs in the vicinity of the PACAP gene locus are associated with schizophrenia.

We also know that Spreds, in contrast, inhibit VEGF-C signaling, which is important in lymphangiogenesis, and that Spred1/Spred2 double knockout (KO) (DKO) mice show abnormal lymphatic development [18].

Our behavioral tests demonstrated that patDp/+ mice show abnormal behaviors.

Two Scn3b−/− mice showed abnormal ventricular rhythms, with one mouse showing abnormalities throughout the 5 min recording period and the second mouse showing such abnormalities after an initial 3 min period of SR.

The resulting mutant mice showed abnormal blood accumulation in the branchial arch region, and arterial abnormalities including a common arterial trunk (failure of the aorta and pulmonary artery to divide) and a highly aneurysmal common aortic sac.

Slc39a13-KO mice showed abnormiceincishowedeth (Figure 2abnormalcclusincisorformiteethnd breakage (3D-mCT, Figure 2A, lower).

In fact, Sprouty2 and Sprouty4 single-deficient mice showed defects of blood vessels rather than lymphatic vessels, while Spred1/Spred2 DKO mice showed abnormal lymphatic vessel development and nearly normal blood vessel development (Fig. 2 and Ref. [18]).

Given the increased expression of anti-inflammatory genes in the striatum and enhanced JNK activity in the substantia nigra of Pink1−/− mice, we were interested to test whether Pink1−/− mice showed abnormal expression of inflammatory and/or anti-inflammatory cytokines in the striatum.