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Homozygote inositol transporter knockout mice show a lithium-like phenotype.
These mice show a marked decrease in NMDA (N-methyl-d-aspartate) glutamate receptor (NMDAR) function.
SIGN-R1-blocked mice show a significant decrease in meningeal neutrophil infiltration.
These mice show a reduction of oxidative stress in several organs, including heart.
In Sox9-conditional-knockout mice, SMG is arrested at the bud stage, although FGF10-knockout mice show a more severe phenotype than Sox9-conditional-knockout mice31.
But if PC-ΔPP2B mice show a relatively normal SS modulation, why then do they not show gain-increase or phase-reversal learning?
Myostatin functions as a negative regulator of skeletal muscle growth and myostatin null mice show a doubling of muscle mass compared to normal mice.
These mice show a significant elevation of serum levels of a C-terminal agrin fragment (CAF) compared to wild-type littermates.
Ets1 deficient mice show a substantial reduction of ILC2s in the bone marrow and lymph nodes.
When exposed to environmental stress (see "Environment: stressors"), DBP knock-out mice show a switch in behavior and become hyperactive.
Data demonstrate that AT(1a) KO mice show a dichotomous response to dehydration, reduced for plasma VP and enhanced for PVN c-Fos protein and VP mRNA.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com