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In addition, both reproductively nonresponsive and long day-housed mice release less GnRH following castration than their intact counterparts.
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On encountering unfamiliar mice, the brains of Asic3−/− ko) mice released less 5-HT than those of Asic3+/+ and Asic3−/−(he) mice.
Indeed, ND/STZ and HFD/STZ mice released less insulin consecutively to glucose stimulation.
In addition, peritoneal macrophages from either DIO or ob/ob mice released less IL-1β in response to LPS compared with control mice.
Moreover, mutant mice that do not have any MCH neurons in the lateral hypothalamus show a reduced preference for sucrose over sucralose, compared to normal mice, and they release less dopamine than normal mice when they consume sucrose.
Supporting this conclusion, islets from pdx1 heterozygous mice released approximately 45% less insulin in response to a glucose stimulus compared to wild-type islets [ 59].
These results indicate that PrPKO mice take up more 59Fe from the intestinal lumen and/or release less into the blood stream compared to Wt controls, resulting in the accumulation of 59Fe in the duodenal epithelium.
Rail accidents are more frequent but generally release less oil.
Well-maintained brakes work more efficiently and release less dust.
AM from TLR2,4−/− mice released a large amount of TNF-α, and KC which was slightly less than that released by WT AM, but not significantly different (Fig. 1).
It was released less than a month after the sinking.
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