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These effects of di-M-PGE2 were much more pronounced in mice receiving differentiated (DMSO-treated) FLC.
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While prostates of mice receiving standard chow were enlarged and burdened with poorly differentiated carcinoma, those of mice on the supplemented diet appeared normal.
While prostates from mice on a standard diet were grossly enlarged and burdened with poorly differentiated carcinoma (74%), those from mice receiving an E/S/L-supplemented diet were normal, both grossly and histologically [ 2, 13].
Instead, in mice receiving OVA323 339-coupled anti-DNGR-1 mAb tOVA323 339-coupledanti-DNGR-1ells differentiated preferentially into IL-17-producing T cells (Fig. 4A and C).
Furthermore, septic mice receiving normal saline experienced significantly more post-sinusoidal adhesion than septic mice receiving any other crystalloid fluid.
Young mice received blood from older mice, and old mice received blood from younger ones.
Then some mice received large doses of quercetin for a week; others received a placebo.
Control mice received saline only.
Control mice received PBS only.
Prior to imaging mice received lucefirin (0.5 mg/mouse, intraperitonially).
Control mice received 177Lu-DOTA-control IgG.
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