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Random forest-based MICE produced more efficient estimates and narrower confidence intervals than parametric MICE, yet in some cases coverage probability was greater than 95%, suggesting that some confidence intervals may be conservative.
Normal mice produced more than four times more melanocytes than did the p53 "knockout" mice.
In 2009, they observed that these "humanized" mice produced more frequent and complex alarm calls, suggesting the human mutations may have been involved in the evolution of more complex speech.
In addition, B6 mice produced more "upward," "short," "one jump," and "more jumps" syllables than BALB mice; whereas BALB mice produced more "flat," "chevron," "complex," and "harmonics" syllables.
In contrast, BALB mice produced more "flat," "chevron," "complex," and "harmonics" syllables (Fig. 4, p<0.05).
In contrast, BALB/c mice produced more "flat," "chevron," "complex," and "harmonics" syllables (p<0.05, t-test).
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Hungry mice produce more of the protein encoded by the SIRT1 gene, the team reports in the 3 March Nature.
In accord with this, AHR ligands suppress TGF-β secretion [38] and the cells of AHR-null mice produce more TGF-β [39].
APP48 mice lack human APP and its metabolites except for Aβ42 whereas APP23 mice produce more Aβ40 than Aβ42 and, in addition to Aβ, N- and C-terminal fragments of APP.
Furthermore, the latency to defection was significantly (P < 0.01) reduced (control: 244.26 ± 29.87 vs. Slc2a8−/�� mice: 113.13 ± 30.18 s) and Slc2a8−/− mice produce more feces during the testing (P < 0.001; Fig. 5d) indicating that arousal of the knockout mice appears to be slightly increased.
We also demonstrate that Tregs from old mice produce more IL-10, downregulate the expression of the costimulatory CD86 on DCs, perturb the redox-mediated microenvironment and cause greater suppression of T-cell proliferation in comparison with Treg from young mice.
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