Sentence examples for mice produced high from inspiring English sources

Tumor-targeted SPION when injected into kininogen-deficient and control mice, produced high levels of vascular clotting in tumors, suggesting that kallikrein activation is not responsible for the nanoparticle-induced thrombosis.

Kupffer cells isolated from ethanol-fed mice produced high amounts of reactive oxygen species and tumor necrosis factor alpha, whereas Kupffer cells from melatonin treatment mice produced less reactive oxygen species and tumor necrosis factor alpha compared with model alcohol-feeding mice.

Mock-vaccinated mice produced high lung titers initially detected at 2 dpi and persisting up to 8 dpi [7.0 and 4.3 log10 50% egg infectious dose (EID50) virus titers, respectively].

Although B cells in NZB dTg mice appeared phenotypically and functionally anergic, these mice produced high serum levels of anti-HEL Ab, suggesting that B cell anergy had been overcome.

All tumors from Hes1 transgenic mice produced high levels of Hes1 but Id1tg tumors A and B had very low levels and tumor C expressed a moderate level of Hes1 (Fig. 7A).

In support of the latter possibility, it is notable that several NZB dTg mice produced high levels of IgA anti-HEL Ab, which is augmented by BAFF, in the absence of significant amounts of IgG anti-HEL Ab, where BAFF plays a relatively minor role (unpublished observation) [45].

Infected cIAP-1 KO mice produced higher pulmonary IFN-γ titers than infected wildtype mice.

In both conditions, peritoneal macrophages obtained from gal3+/+ mice produced higher NO levels (Figure 6C), a response that was reproduced by cells stimulated with LPS (positive control).

This hypothesis is supported by the observation that microglial cells isolated from MPSIIIB mice produced higher amounts of MIP1α and IL1ß mRNAs in vitro in response to LPS or HS oligosaccharides than wild type mouse microglia.

After 48 hours of culture, we found that splenocytes from HIVBr18 immunized mice produced higher levels of type I cytokines like IFNγ, IL-2 and TNFα and negligible levels of IL-5 and IL-4.

Macrophages from diabetic mice produced higher levels of the pro-inflammatory cytokines TNF-α & IL-6 and lower anti-inflammatory cytokine IL-10 compared to non-diabetic controls (Fig. 5B).