Sentence examples for mice previously reported from inspiring English sources

First, transgenic mice we generated display a high-bone-mass phenotype, whereas transgenic mice previously reported show an osteopenic phenotype.

Comparison of our results with the phenotypes of XBP1 KO mice previously reported indicates the possibilities that IRE1α has functions independent of XBP1 and XBP1 has functions independent of IRE1α.

Second, transgenic mice previously reported are smaller in size and have a higher rate of postnatal mortality, whereas transgenic mice we generated are fertile and normal in appearance and body weight.

A possible explanation for these discrepancies is that transgenic mice we generated overexpress Cthrc1 specifically in osteoblasts under the control of the Col1a1 2.3kb osteoblast-specific promoter to focus on the function of Cthrc1 in bones, while transgenic mice previously reported ubiquitously overexpress Cthrc1 under the control of cytomegalovirus promoter.

We also found reduced photoreceptor responses in Adam9 knockout mice, previously reported to be asymptomatic.

This is similar to the time course for infection in C57BL/6 mice previously reported in the literature [ 30, 31].

Cdc42 cardiomyocyte knockout mice were generated by crossing Cdc42/flox mice with MLC-2a Cre mice, as previously reported [22].

We found that sclerostin was expressed in the joint cartilage of mice, as previously reported in sheep, mice and humans [ 15], although it was restricted to calcified cartilage.

There was no significant correlation between the gene expression profile for Sec24a gt/gt mice and previously reported profiles from mice with altered expression of SCAP or SREBP1/2 (Horton et al., 2003).

For the genetic analysis of VRK1 in mice, we used VRK1-deficient mice as previously reported (Figure S1) [17].

When we compared the depolarization-induced glutamate release in Balb/c mice with previously reported values for the C57BL/6 Glud1 mice (Bao et al., 2009), an interesting trend emerged.