Sentence examples for mice presented an from inspiring English sources

In this sense, microarray data evidenced that ob/ob mice presented an upregulation of proteolytic (Need4, Ube2h), apoptotic (Acin, Amid), and autophagic (Lysmd3, Becn1, Atg121) genes, which were down-regulated by leptin treatment.

Besides, we found that 30.8% of the cells obtained from MLNs of galectin-3−/− mice presented an abnormal DNA content, suggesting hyperploidy or the presence of a large amount of aggregates of nuclei derived from dead cells (Figure 6A and 6B, in M5).

Furthermore, TgR mice presented an extreme sensitivity to cholinergic stimulators.

TgCRND8 mice presented an increased LTD, which was reverted by D-JNKI1 treatment in 9-months-old AD animals.

ALDHhiSSCloVLA4+ NSC-treated SOD1 mice presented an amelioration of the motor neuron phenotype, demonstrated by neuromuscular function tests and increased survival.

Intriguingly, however, these mice presented an increased rate of ataxia because of mononuclear infiltration into the cerebellum instead of spinal cord.

Interestingly, fasted N2KO mice presented a significant, 5.16-fold (*p≤0.05, T value = −2.99, df = 4) increase in serum leptin levels compared to WT mice.

Wild-type mice presented a significant [F 5) = 54.810; P<0.01] increase in the response to the 2nd pulse at the 40-ms interval (Fig. 3A).

In contrast, RASSF9−/− mice presented a dramatic change in epithelial organization of skin with increased proliferation and aberrant differentiation as detected by bromodeoxyuridine incorporation assays and immunofluorescence analyses.

Miura and co-workers have previously shown that 2-months old mice overexpressing PGC-1α ran less than wild-type mice because these transgenic mice presented a mitochondrial uncoupling and a marked decrease in the ATP muscle content [26].

In accordance with previous descriptions [24] and with present results (see arrow in Fig. 2B), both wild-type and TgIE96 mice presented a noticeable alpha response to the tone used as a CS.