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As control, an iPSC clone from wild-type mice of identical background was also obtained which will be referred to as wt-iPSC.
Here, we compared HO-1 expression in SCD and control mice of identical genetic backgrounds, in both the Townes and Berkeley models (Figure 5, 6, 7).
Two mice of identical genotypes, which were previously housed in different cages, were placed into a box together (175×245×125 mm) and allowed to explore freely for 10 min. Social behavior was monitored by a video camera.
Data were collected for 120 min. In the social interaction test, two mice of identical genotypes that were previously housed in different cages were placed in a box together (40×40×30 cm) and allowed to explore freely for 10 min [15].
For social interaction test in a novel environment two mice of identical genotypes that were previously housed in different cages, were placed into a box together (40×40×30 cm) and allowed to explore freely for 10 min. Social behavior was monitored by a CCD camera.
In the social interaction test, two mice of identical genotypes that were previously housed in different cages were placed in a box together (40×40×30 cm) and allowed to explore freely for 10 min. Social behavior was monitored with a CCD camera.
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Liver cirrhosis was induced by thioacetamide in wild type and hSDC1+/+ mice of the identical strain.
Control groups of mice received identical quantities of plasmid-free bacteria NZ9000 or PBS.
In this study, we closely monitored ARDS in mice, caused by inoculation of identical doses of 2 different influenza strains rendered highly pathogenic toward mice by adaptation.
Despite these deficiencies, the risk of bias introduced by non-random division of identical mice (gender, age, mass, genetics) is low.
Our general approach was to seed adult and old mice with equal numbers of identical adult OT-I TCR transgenic CD8 T cells and to compare their ability to expand in age-disparate recipient environments in response to microbial challenge.
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