As mentioned previously, numerous IL-21+ cells were detected within and near the cortex in stroke; they might contribute to the fate of neurons during disease progression, as recently proposed for IL-17 in neurons in humans and mice, where it correlates directly with neurotoxicity.
P16 is induced in mouse neoplasia, where it correlates with OIS (Burd et al., 2013; Kuilman et al., 2010).
In our VMD2-IL-10 transgenic mice, it appears that disease progression correlated with infiltration of macrophages into the sciatic nerve and subsequent demyelination.
In this study, we compared FDG uptake in mammary tumors driven by the Akt1, c-MYC, HER2/neu, Wnt1, or H-Ras oncogenes in genetically engineered mice, correlating it to tumor growth, cell proliferation, and expression levels of gene involved in key steps of glycolytic metabolism.
One of the most popular experimental designs has been to determine the gene expression changes that occur in knockout mice where it is correlated with phenotypic changes.
The amount of IL-1β was reproducible between mice and correlates with the ELISA data.
Therefore, our results suggest that there is an absent of stress-induced oxytocinergic activation in Per1 mutant mice, and it could be correlated with the deficiency of stress induced antinociception in this strain.
There was an increase in plasma MT levels in tumour-bearing mice and it was positively correlated with the increase in hepatic MT levels.
In this study, we showed that the over-representation of Eno2 in the cerebellum of mutant mice correlates with a significant increase in its serum levels.
It has been reported that the plasma concentration of erlotinib in athymic nude mice correlates well with erlotinib concentration in tumor.
