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The long-term survival frequency of Sulf1−/− Sulf2−/− mice is therefore ∼15%.
Physiological characterization of this model in addition to APPYG/YG mice is therefore of great interest.
ECTV infection of mice is therefore uniquely able to provide physiologically relevant insights into co-evolved host-pathogen relationships.
This strain of mice is therefore ideal for studying the role of DNA repair enzymes and Bax in the spontaneous induction of apoptosis.
Activation of the prelimbic cortex in R mice is therefore consistent with a role for this component of the mPFC in drug-primed reinstatement behaviour.
The gene expression profile of stromal cells associated with tumor progression in CR2-TAg mice is therefore consistent with ECM remodeling functions that may be expected in a robust stromal reaction to processes ranging from mechanical tissue injury to tumor growth.
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I couldn't help but feel a tension between the claims that the mouse genome is far more similar to the human genome than scientists previously believed and that mice are therefore an excellent surrogate for studying the human genome in tests that would be unethical to conduct on human beings.
HHD mice are therefore useful for studying immunisation strategies for inducing and maintaining CD8 responses to over-expressed self antigens.
RO5126766 treatment (1.0 mg/kg) did not inhibit growth of the COLO320DM tumors (Figure 2a) and these mice were therefore sacrificed after 6 days of treatment when the tumors had reached the size limits allowed by research ethics.
All experiments involving doubly mutant mice were therefore performed on F1 mice.
These mice are therefore a valuable tool in examining the pathological cellular mechanisms underlying seizure genesis in SSADH deficiency.
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