Sentence examples for mice is probably from inspiring English sources

Thus, the attenuation of DAMGO-induced antinociception in streptozotocin-induced diabetic mice is probably caused by dysfunction in cellular pathways after the activation of G-proteins.

The observation that tumour outgrowth was not evident in all mice is probably related to incomplete TRM depletion, protection by non-depleted endogenous TRM cells, and/or melanoma elimination in some mice before TRM depletion.

This very restricted spatial and temporal pattern of expression of this line of Tet-ΔGR/EGFP transgenic mice is probably due to the site of integration of the transgene within the genome.

The slower mobilization of glycogen stores in transgenic compared to control mice is probably due to plasma insulin, which remained high during fasting, and to the preference for lipid oxidation over glucose oxidation.

Thus, CCR2 plays a role in the pathogenesis of severe experimental dengue infection and it appears that enhanced survival in CCR2 /– mice is probably secondary to decreased liver damage, decreased cell activation and decreased cytokine storm.

These results demonstrate that propofol can induce direct tau hyperphosphorylation in neuronal cells, and suggest that, as propofol can cross the blood-brain barrier [29], the outcome observed in mice is probably due to a direct pharmacologic effect on neurons.

A practical problem is that the writers will not be able to have their exterminator treat the common areas of the building, like the basement, where the mice are probably coming from, Mr. Kaminsky said.

Increased T cell proliferation in vitro or increased expansion of T cells in vivo in HLA-DR3.IFN-γ−/− mice was probably not due to increased expression of HLA-DR3 molecules in HLA-DR3.IFN-γ−/− mice because of comparable numbers of CD4+ T cells were present in both groups of mice in both thymus and spleen (Fig. 7).

Reduced ET-1 levels in Fn14−/− mice are probably due to reduced fibrosis [ 10, 29].

As such, the short lifespan of Tsc1c/− /hGFAP2- Cre+ mutant mice was probably a consequence of the onset of status epilepticus, rather than of cachexia and wasting.

Since insulin is the initiating autoantigen in the NOD mouse, but other autoantigens such as GAD65 are recognised as the disease progresses [ 24], protection after B 9-23/IFA immunisation in older NOD mice was probably also mediated by bystander suppression mechanisms [ 25– 28].