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Further, the lifespan of mdx mice is only moderately reduced [2], [3].

Although the embryonic outgrowth of mammary rudiments is Wnt-dependent [15], [16], the phenotype of early mammary development of Lrp5−/− mice is only marginally affected.

This latter possibility is supported by our previous study, which indicated that the systemic blood pressure in BK channel-ablated mice is only moderately increased [27].

Although this prnp genotype of TgOvPrP4 mice is only rarely identified in sheep with atypical scrapie [13] [16], [26], [30] [33], most of the inoculated mice accumulated the pathological prion protein in their brain.

Indeed recent studies found [88], [89] that the cochleotopic mapping in primary auditory cortex of mice is only present on a (relatively) large scale, whereas local neuronal populations show less organized cochleotopic gradients.

This result is consistent with the finding by Takizawa et al that decreased Ig titers in immunized AID+/− mice is only detected for antigen-specific high affinity antibodies [36].

Despite high RTB-specific serum antibody titers, groups of RTB immunized mice were only partially immune to ricin challenge.

According to this scenario, the 'CB3' receptor is only partially responsible for newborn milk suckling since the cannabinoid CB1 knockout mice were only partially affected by the antagonist.

Interestingly, the Cox1 null mice generally had no identifiable gastrointestinal abnormality, despite the fact that gastric PGE2 levels in null mice were only 1% of the levels observed in wild type mice (Langenbach et al., 1995).

The other two groups of mice were only treated with L435-3 or mock control.

Until recently, quantification of myocardial infarct size in mice was only possible by use of post-mortem histological analysis.