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The increased heart rate leading to a positive inotropic effect in the hearts of transgenic mice is, at least in part, due to the overexpression of adrenergic receptors.
We conclude that the increased tumor growth observed in adiponectin deficient mice is at least partly due to the reduction of macrophage infiltration and not by enhanced angiogenesis.
These findings suggest that decreased Abeta level in these mice is at least partly attributed to S14G-HN-induced increase of neprilysin level.
Since we find that one of the primary sites of dysfunction in Cra1/+ mice is at the NMJ, variations among laboratories in housing conditions and daily food intake will impact the emergence of this phenotype and may account for some of the divergent reports of phenotypes.
The indicated age of mice is at the day of the terminal optical imaging experiment.
The onset of Golgi fragmentation in G1del mice is at about 14 15 weeks of age [ 29].
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When infected humans or mice are at their sickest, they're already well on their way to eliminating the microbes that cause the disease.
Here we show that a large fraction of medial entorhinal cortex neurons fire specifically when mice are at given distances and directions from spatially confined objects.
However, recent results using this model in adult mice are at odds with the same model in neonatal rats.
After CsA administration, the peak brain uptake in mice was at 1.5 min and at 1.2 min in rats.
The first detected TCR expression in Rec-HY mice was at the DN4 stage of development.
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