Sentence examples for mice induced a from inspiring English sources

Furthermore, the transfer of CD4+CD25+ T cells from EPIT-treated C57BL/6 mice induced a significant decrease in the peanut-induced production of Th2 cytokines (i.e. IL-5 and IL-13, P < 0.05), which was as efficient as the EPIT treatment of WT mice (Fig. 8a).

Interestingly, metacercariae challenge of resistant rIL-25-treated mice induced a peak of IL-12p35, theugh the presence of IL-25 prevented the development of a Th1 phenotype as it occurs in primary infections in naïve mice.

First, we showed that dorsal hippocampal but not medial prefrontal cortex lesions in C57Bl6 mice induced a time-dependent impairment of spatial recognition.

More importantly, transfer of IL-35Ig−transfected, IGRP-pulsed DCs into prediabetic NOD mice induced a delayed and less severe form of diabetes, an effect accompanied by the increase of CD4+CD39+ suppressive T cells in pancreatic lymph nodes.

We therefore hypothesized that eNOS overexpression could restore both the impaired neovascularization response and decreased proangiogenic function of BMMNCs in clinically relevant models of diabetes and hypercholesterolemia. Transgenic eNOS overexpression in diabetic, atherosclerotic, and wild-type mice induced a 1.5- to 2.3-fold increase in postischemic neovascularization compared with control.

It has been previously shown that TRα gene invalidation in mice induced a significant decrease in body temperature [21].

Intranasal administration of these recombinant lactococci in mice induced an HPV-16 E7-specific immune response.

Similar to our results, Citrobacter rodentium infection in mice induced an intestinal Th1 environment with simultaneous classical and alternative activation of macrophages37.

However, administration of SEA to these same mice induced an initial episode of EAE which was characterized by severe hindleg paralysis and accelerated onset of disease.

When CD40 was engaged for 20 hrs, both populations from NOD, BALB/c, and NOR mice induced an increase in CD40 expression demonstrating a positive feedback loop.

Taken together, this indicated that HSP70 immunization of IL-10−/− mice induced an HSP70-specific B and T cell response that, in the absence of IL-10, failed to suppress PGIA development and PG-specific pathogenic IgG2a production.