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Individual wild mice have unique expression patterns of Mups in their urine [24], [25].
This study clearly shows that t-haplotype mice have unique and characteristic Prdm9 ZFA and intronic (HSR1) sequences.
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Given that most data were obtained on selected inbred strains of mice that have unique phenotypes and requirements for disease development, understanding human lupus is even more challenging.
Similarly, 5WexGF mice may have unique characteristics with respect to the IFN-α response, which is critically important not only for protection against infection by virus and certain microorganism, but also for control of autoimmune responses.
We note that individuals from each of these three populations (yeast, mice, and humans) have unique genetic histories, and our analysis suggests that this influences the number and type of eQTL detected in each study.
Individual mice have a unique odor, or odortype, that facilitates individual recognition.
Our GAD67-GFP BAC transgenic mice provide another genetic tool for studying adult neurogenesis, and these mice have several unique features.
Microarray analysis revealed that β2KO mice have a unique genetic profile both before and after ischaemia.
However, DNase 1L2-deficient mice have a unique phenotype, with undigested nuclei being present only in hair shaft and nail plate.
In summary (Fig. 9), these data indicate that all Tg and KO mice with disrupted collagen X function have unique skeletal and hematopoietic defects that arise from collagen X disruption in hypertrophic cartilage.
The functions of these receptors in TNFα signaling continues to be investigated [ 30], although recent evidence from TNF receptor knockout mice suggests that both TNF receptors have unique contributions to spinal cord synaptic plasticity and inflammatory pain [ 31].
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