Sentence examples for mice has identified from inspiring English sources

The use of genetically-engineered mice has identified α2- and α3-subunit containing GABAA receptors as principal contributors to the spinal disinhibition that occurs after inflammation and neuropathic injury.

Genetic dissection in mice has identified the importance of several members of the innate immune system in the so-called priming phase of PH, including IL-6 and members of the complement system.

Work with PAR2-null mice has identified PAR2 as a potential therapeutic target for arthritis [ 103].

Much research, based on transmission characteristics to inbred mice, has identified more than 15 classical scrapie isolates with distinct pathologies [ 12].

Indeed, the development of osteopetrosis in a variety of knockout mice has identified necessary functions of genes in osteoclast biology that largely had not been anticipated [ 3, 4].

A genetic screen for epigenetic modifiers in mice has identified the SmcHD1 gene, which is required for maintaining DNA methylation and repression of Xi-linked gene promoters (Blewitt et al., 2008).

Genetic engineering studies in mice have identified a number of subunits that are critical for the ability of nicotine to activate the reward system in the brain, consisting of the dopaminergic cell bodies in the ventral tegmental area and their terminals in the nucleus accumbens and other portions of the mesolimbic system.

Studies of homozygous shiverer mutant mice have identified ATF2 as a regulator of the expression of MAG expression at a specific stage of shi/she oligodendrocyte differentiation [270].

Detailed immunohistochemical studies in mice have identified highly regulated, specific domains of discrete and dynamic ERK phosphorylation throughout development, including the pharyngeal arches and limb buds [26].

Multiple studies of adiposity and hypertension in genetic crosses from rats and mice have identified a large number of QTL associated with these traits [13] [18].

Further studies in mice have identified some transcriptional targets of PPARγ in trophoblast [10] and shown that treatment of pregnant mothers with PPARγ agonists led to fetal and placental growth restriction in a PPARγ-dependent manner [11].