They saw none of the biomarkers associated with lung inflammation or cellular damage from metal nanoparticles, and the mice had normal breathing.
Correspondingly, CA IX deficient mice had normal gastric pH, acid secretion, and serum gastrin levels.
And the histopathological analysis revealed that SPG treated mice had normal liver architecture and no necrosis.
mCD40-LMP1tg mice had normal lymphocyte subsets, and immunization elicited an antibody response featuring normal isotype switching, affinity maturation, and germinal center (GC) formation.
Our results demonstrated that while COX-2−/− female mice had normal bone geometry and trabecular microarchitecture at 4 months of age, the male knockout mice displayed reduced bone volume fraction within the distal femoral metaphysis.
These results suggested that female mutant mice had normal folliculogenesis.
NOS2 mutant mice had normal reflexes and no apparent motor deficit.
N1N2K5 TSLPR−/− mice had normal sized spleens and lymph nodes (Figure 5C).
Tnnt2+/− mice had normal hearts (histology, mass, left ventricular end diastolic diameter [LVEDD], fractional shortening [FS]).
Accordingly, GLUT2-loop transgenic mice had normal insulin sensitivity in peripheral tissues, mainly muscle and adipose tissue.
Although the retina of the mutant mice had normal architecture, the number of apoptotic cells was increased.
