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The untreated mice group was considered as control.
A SS mice group was pretreated with HO-1-specific inhibitor (ZnPP IX) or a specific inhibitor of sGC (ODQ).
In line with our previous results, tumor outgrowth in naive Nr2f6−/− mice group was significantly delayed even with this high tumor load.
The A β plaque area in the EB101 immunized mice group was reduced by 29.7 34.5%, relative to the other tested groups.
However, when the colitis-induced mice group was fed with Lf1, the downregulation was considerably reduced to −37.037-fold indicating that there was actually a substantial increase (approximately 63%) in the expression of " SOD2" gene under these conditions.
The survivability rate of the ACF treated mice group was 100% and that of CQ treated mice was 75%, whereas only 25% survivability rate was observed in untreated control mice under the same experimental conditions.
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The cDNA probes from the tissue of control group were labeled with dCTP-Cy5, while those from the infected mice group were labeled with dCTP-Cy3.
The magnitude of the peak parasitemias between the different mice groups was not significantly different when comparing groups of mice infected with 105 or 104.
The hair morphogenesis stage of each hair follicle in each mouse group was evaluated as described previously [13].
The hair cycle stage of each hair follicle in each mouse group was evaluated as described previously [27].
One mouse group was examined 24 hours after stopping exposure.
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