Sentence examples for mice from AD-like from inspiring English sources

Similarly, IGF1 signaling reduction protects mice from AD-like disease.

Further evidence for transmissibility emerges from APP transgenic models in which mice develop AD-like pathology only late in life ([ 156] for review).

Results: HO-1 expression is increased in GFAP.HMOX1 mice relative to wild type and in 11 12-month-old 11 12-month-old 11 12-month-oldotype) relative to control mice and 5–6-month-old 3xTg-AD mice (no AD-like phenotype).

In this context, we treated mice with AD-like pathologies with VC.

Here, we show that phospholipase C-β3 (PLC-β3 -deficient mice sPLC-β3 -deficientloPLC-β3 -deficientsions and mice spontaneouslyen-indevelopermatitis than wild-type mice.

By contrast, EE has no benefits on cognition and neurogenesis in transgenic mice with an AD-like aggressive phenotype [38].

The present study demonstrated that PLC-β3-deficient mice spontaneously develop AD-like skin lesions.

We next evaluated hippocampal memory in 3 months old APP/PS1 mice that lack AD-like pathology.

Following repeated application of DNCB to the back skin of mice for 2 weeks, AD-like lesions developed.

Nc/Nga mice are characterized by AD-like skin lesions and show elevated levels of blood IgE [ 27].

In transgenic mice with early-stage AD-like pathology, Lu AF20513 produced Aβ antibodies and induced robust nonself T-cell responses that reduced AD-like pathology without inducing microglial activation and enhancing astrocytosis or cerebral amyloid angiopathy [ 38].