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The high-fat fed EPO transfected mice appeared noticeably leaner than control mice fed the same diet.
Mice on this diet grew more slowly (14%) than mice fed the HF−F diet.
Mice (6 weeks old) were divided into four groups: (1) WT mice fed the basal diet (n = 12 mice), (2) KO mice fed the basal diet (n = 11), (3) WT mice fed the high-fat diet (n = 11), and (4) KO mice fed the high-fat diet (n = 11).
Mice (6 weeks old) were divided into four groups: (1) WT mice fed the basal diet (n = 10), (2) KO mice fed the basal diet (n = 10), (3) WT mice fed the high-fat diet (n = 10), and (4) KO mice fed the high-fat diet (n = 10).
Mice fed with a TLE rich diet and exposed to DSS lost more weight than mice fed the control diet.
Though his mice did not lose weight, they lived far longer than the undosed mice fed the same diet.
The liver steatosis observed in mice fed the HF diet was also prevented by PCLA.
In mice fed the MCD diet for 1 week, weak steatosis, but no inflammation, was observed in the liver.
Figure 2 Histochemical investigation from the liver of mice fed the normal or MCD diet for 2 or 4 weeks.
An increase in liver weight in mice fed the purified EPS was observed, but with no change in liver lipids.
The production of RANTES and JE/MCP-1 by spleen cells in mice fed the FO diets was significantly lower than in mice fed the CO diets (p < 0.0001).
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