Sentence examples for mice expressed increased from inspiring English sources

The reconstituting B cell repertoire in wild type mice contained an increased frequency of DNA-reactive B cells; in heavy chain transgenic mice, the reconstituting repertoire was characterized by an increased frequency of mature, high affinity DNA-reactive B cells and the mice expressed increased levels of serum anti-DNA antibodies.

These results indicate that TH mice expressed increased levels of the osteoclastogenic markers RANKL and IL-6 and reduced amounts of OCN and OPG in bone marrow, resulting in significant bone density loss.

GW501516-treated wild-type mice expressed increased levels of PDK1, pT308AKT and pS9GSK3β, whereas, transgenic mice exhibited increased pT308AKT, but no additional changes in PDK1 and pS9GSK3β, a result that we attribute to their already elevated levels in the absence of GW501516 treatment.

Non-stimulated splenic MDSCs from 4T1 cell-bearing mice expressed increased levels of both Adam10 and Adam17 compared to MDSCs from EMT6 cell-bearing mice and naïve mice.

The CD44−CD25low cells from LckCre+LKB1fl/fl mice expressed increased levels of Hes-1 and Deltex1 when compared with control cells (Fig. 3E).

In addition to increased HA accumulation, HYAL-deficient mice expressed increased levels of CD44, α-SMA, and collagen compared to wild-type mice 30 days after renal injury [ 66].

Altogether, these data demonstrate that ΔCS mutant mice express increased levels of mFasL and no detectable sFasL.

In mice expressing increased levels of GATA-6 in respiratory epithelial cells, postnatal alveolarization was disrupted, resulting in airspace enlargement.

Others have shown that TLR4−/− mice express increased levels of superoxide in isolated lung endothelial cells which leads to emphysema as the mice age [5].

Notably, however, mice expressing increased levels of STIP1 show no major phenotype when examined using a range of behavioral tasks, whereas mice expressing reduced levels of STIP1 exhibit attention deficits and are hyperactive.

The amplification of certain cell types in these mutants can be related to the phenotypes observed in mice expressing increased levels of the oncogene c-MYC, which causes transcriptional amplification of pre-existing transcriptional programs rather than targeting specific genes, resulting in a variety of tumorous cell types (Lin et al., 2012).