Sentence examples for mice expressed higher from inspiring English sources

Indeed, the bones from Snail1-ER tamoxifen-treated mice expressed higher levels of Collagen I transcripts and lower levels of Osteocalcin expression in Snail1-ER bones, markers of early and late osteoblast differentiation, respectively (Nakashima and de Crombrugghe, 2003), while the number of osteoblasts remained similar.

Finally, Tfh cells from PbAWT-infected mice expressed higher levels of the cytokine IFN-γ (Supplementary Figure 2d).

In addition, TTC9A−/− OVX mice expressed higher levels of TPH1, TPH2 and 5-HTT in vmPFC than TTC9A+/+ OVX mice.

Tregs from Krt76−/− mice expressed higher levels of the Treg suppressive markers CD39 and CD73 compared to control Tregs (Fig. 4e, f).

The Tg6F-fed mice expressed higher levels of IL-4, IL-10, IL-12, TLR2, IFNβ1, MX1 and NFKBID compared to chow-fed mice in both jejunum and lung (Fig. 2a,b).

However, after peripheral LPS treatment Pink1−/− mice expressed higher levels of striatal IL-1β, IL-10 and IL-12 than wildtype controls.

Whereas BMDCs and a subset of myeloid DCs from hCD1Tg mice expressed high levels of CD1b, the expression of CD1b was almost undetectable on bone marrow derived macrophages (BMDMs), similar to the observation in human monocyte derived macrophages.

Interestingly, the BCR-ABL1-expressing mast cells in the secondary mice expressed high levels of CD25 (IL2Ra), a cell surface receptor known to be expressed on neoplastic mast cells.

CD11b(low)CD11c(high) DC from FL-treated mice expressed high levels of intracellular MHC class II.

DC from both pGM-CSF and FL-treated mice expressed high levels of surface class II, low levels of the costimulatory molecules CD40, CD80, and CD86 and were equally efficient at stimulating allogeneic and Ag-specific T cell proliferation in vitro.

Next, we investigated the effects of EPA on macrophages in vivo, and demonstrated that ATMs of EPA-fed mice expressed high levels of CD206, a marker of M2 macrophages, indicating that EPA induces polarization of ATMs into anti-inflammatory M2 phenotype.