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Conversely, pulmonary homogenates of non-protected mice expressed a predominantly Th-2 type cytokine profile.
Furthermore, B6 mice expressed a socially conditioned place preference (SCPP) throughout early and late adolescence (Fig. 10b).
Here, we demonstrated that B1B2KO mice expressed a pro-inflammatory phenotype that might explain part of their renal dysfunction.
However, uko/3cv mice expressed a slightly shortened dystrophin protein at the same level as that of 3cv mice.
3cv mice expressed a near full-length dystrophin protein at approximately 5% of the normal level (Figure 2) [7], [7].
Because Mecr transgenic mice expressed a phenotype with some features of dilated cardiomyopathies, we challenged the mice to exercise on a treadmill.
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The vast majority of B cells of HIS mice expressed an IgM (97.9±1.0%), whereas IgG (1.8±1.0%) and IgA (0.07±0.04%) expressing B cells represented minor populations.
In that study, LKB1 floxed mice were crossed to mice expressing a RIP2-promoter driving Cre-recombinase expression.
Mice heterozygous for the conditional allele (Pik3c3flox/+) [32], [32] were bred with mice expressing a Meox-cre transgene with cre expressed in the germ line to generate Pik3c3+/− animals.
In mice expressing a single TCR specific for a non-self-antigen, no Treg develop.
These mice express a nonmuscle isoform of the myosin light chain kinase gene (nmMLCK2) targeted to the endothelium.
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