The Vβ12-transgenic mouse expresses several related but distinct T-cell clones specific for the galactosylated CII peptide.
Additionally, we and others found that rats and mice differentially express several other pain-relevant genes in peptidergic and nonpeptidergic nociceptive neurons, including the noxious heat receptor TRPV1 and Mas-related G protein-coupled receptors [35], [36], [37].
This change in cell fate is thought to occur as mouse PGCs already express several pluripotency markers and respond to growth factors present in the culture medium [8].
Probing this further, Abner Notkins and his colleagues at the National Institute of Dental Research in Bethesda, Maryland, studied the effects of pregnancy hormones on a mouse strain engineered to express several HIV-1 genes.
While many mouse neuroblastoma cell lines can express several neuronal properties, other lines lack specialized neural functions and may re-express embryonal or foetal antigens, suggesting some reversion towards an earlier stage of differentiation.
Mouse ES cells exhibit high levels of autocrine Smad2/3 signalling and express several TGFβ signalling factors including Nodal [19].
Previous studies show that this gene expresses several splice variants in human, mouse, and dog.
EmNCSCs expressed several transcription factors characteristic of early NC specification in chick, mouse, and Xenopus.
After induction, isolated CD31+ cells expressed several endothelial cell marker genes and exhibited the ability to form vascular network structures similar to CD31+ cells from neonatal mouse heart.
Mice expressing the unedited serotonin receptors developed normally, but mice expressing the edited version presented with several abnormalities, including growth retardation and hyperphagia.
