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These mice express a nonmuscle isoform of the myosin light chain kinase gene (nmMLCK2) targeted to the endothelium.
Immunodeficient mice express a naturally mutated form of BAFF-R [24].
These mice express a transgenic recombinase under the control of the Villin-Cre promoter.
Our ApoE*3Leiden.CETP mice express a natural mutation of the human APOE3 gene in addition to the human APOC1 gene.
These mice express a membrane-bound form of OVA that is expressed under the control of β-actin promoter.
Briefly, RenTg mice express a renin transgene inserted into a liver-specific locus and driven by a liver-specific promoter/enhancer.
The ΔCS mFasL mice express a gene-targeted form of FasL in which the major cleavage sites were mutated to render the molecule resistant to metalloproteinases.
These mice express a fosGFP fusion protein and have been used to identify and characterize recently activated neurons in live brain tissue [21].
It is well established that mdx3cv (3cv) mice express a near-full length dystrophin protein at ∼5% of the normal level.
In this model, the mice express a TCR specific for LCMV gp33 41 peptide and the LCMV gp protein expressed in β-pancreatic cells.
VC+ mice express a transgenic recombinase under the control of the Villin-Cre promoter that causes an IEC-specific deletion of PPAR γ.
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