Sentence examples for mice exhibiting abnormal from inspiring English sources

In many mice exhibiting abnormal circadian behavioral rhythms, the expression of clock genes is severely affected in the SCN [15], [38] [44].

Our goal was to define genes and pathways that were altered in α9−/− mice exhibiting abnormal innervation patterns during development as a consequence of gene ablation restricted in space (in the cochlea) to the postsynaptic hair cells, which represent the target of the descending nicotinic efferent innervation.

Compared with control, tgMT mice exhibited abnormal gait, as evidenced by increased forelimb and hindlimb stride lengths, suggesting abnormalities of upper motor neurons in tgMT mice.

Li, Q. et al. IKK1-deficient mice exhibit abnormal development of skin and skeleton.

Keratinocytes from these mice exhibited abnormal expression of adhesion molecules at both the protein and the mRNA levels.

MafB-deficient mice exhibited abnormal podocyte and macrophage differentiation, suggesting ancestral and functional overlap.

IKKα deficient mice exhibit abnormal morphogenesis and developmental defects [17] [19].

The γ/ε-fc mice exhibit abnormal endplate distribution, axonal growth, muscle innervation, NMJ formation and distribution, as well as endplate morphology.

We have previously reported that transgenic virgin mice over-expressing the human heparanase gene (hpa-tg mice) exhibited abnormal abundant branching of ducts in the mammary gland and precocious alveolar structures, typical of pregnant mice [36].

Bst+/− mice exhibit abnormal PLR, but have preserved circadian rhythms.

Physiologically, Per2-mutant mice exhibit abnormal long-term potentiation.