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By contrast, wild-type mice exhibited weaker forms of the fixed action pattern, and often failed to complete the full sequence.
In the spinal cord (E11) (Fig. 7E G) and cerebellum (E14) (Fig. 7H M), Gadd45g-d4Venus mice enabled very intense visualization of differentiating cells, including motor neurons, whereas Neurog2-d4Venus mice exhibited weaker fluorescence.
Histological analysis indicated that surviving LGR5+ cells in 9-month-old G3 mTerc−/− mice exhibited weaker GFP staining intensity and were mostly located at position 4 above the Paneth cells, whereas LGR5+ cells at the crypt base that were located in between the Paneth cells (position 1 and 2) were preferentially depleted (Fig 3K O).
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Globally, mice exhibited weak spatial selectivity as considering the target quadrant, or the target platform zone.
Since non-vaccinated wild-type mice exhibited weak endogenous anti-tumor T cell activity by CTL assay, and such activity was substantially increased upon vaccination (Fig. 2C), this suggested that stem-like gene expression in gliomas is modulated in direct proportion to anti-tumor CTL activity levels.
Furthermore, compared to the Osx Cre control mice, the Osx:EfnB1−/− mice exhibited significantly weaker and less rigid bones, with a reduction in trabecular/ cortical bone formation, reduced trabecular architecture and a reduction in the size of the growth plates at the distal end of the femora from newborn through to 4 weeks of age.
HDC−/− mice following sepsis induction exhibited weaker NGAL staining in kidneys.
Similar to our observations with untreated mice, Hoxa5 −/− mice exhibited a weak CC10 immunoreactivity when compared to controls (Fig. 3, Fig. 5G L).
Moreover, a viewpoint designed to a mouse-divergent site exhibited weak interactions within the mouse domain, analogous to the local insulation behavior observed in Figure 3B.
They exhibited weaker NOEs to C H6.
Similarly, Brca2-deficient mouse embryonic fibroblasts exhibited weak SAC activity.
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