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Similar results occurred with the arthritogenic alphavirus Ross River Fever virus in which complement-deficient mice exhibited far less severe disease signs and tissue damage than WT mice despite similar viral titers [24], [31], [32].
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Krt76−/− mice exhibited deregulated cytokine expression.
miR-223−/+ mice exhibited more severe ALI and cytokine dysregulation.
Our results demonstrated that TTC9A−/− mice exhibited an increase in anxiety-like behaviors compared to the wild type TTC9A+/+ mice.
In addition, the BLM-injected Ninj1 KO mice exhibited a mild fibrotic phenotype, as compared to WT mice.
You probably won't be surprised to learn that the campylobacter-infected mice exhibited more anxiety when navigating a maze than the control mice.
QPRT+/− mice exhibited higher quinolinate, lower NAD+, and higher AKI susceptibility.
In our studies, control mice exhibited robust rhythms in ECG parameters including RR, PR and QTc.
AhR−/− mice exhibited rapid body weight loss leading to termination of the experiment at 60 h.
We also found that Dio2-knockout mice exhibited enhanced bleomycin-induced lung fibrosis.
Conversely, ERRγ+/− mice exhibited lower exercise capacity compared with their wild-type littermates.
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