In addition, the BLM-injected Ninj1 KO mice exhibited a mild fibrotic phenotype, as compared to WT mice.
JZL184-treated mice exhibited a broad array of CB1-dependent behavioral effects, including analgesia, hypothermia and hypomotility.
Glomus cells from partially HIF-1α deficient mice exhibited a normal sensitivity to hypoxia.
Knockout mice exhibited a dramatic 85% decline in intramuscular NAD content, accompanied by fiber degeneration and progressive loss of both muscle strength and treadmill endurance.
ApoE knockout mice exhibited a similar synaptic activity profile with apoE4 TR mice at 7 months.
On MRI, the mice exhibited a median of four metastases on day 20 (range = 3 11).
Finally, synapsin I-deficient mice exhibited a strikingly increased response to electrical stimulation, as measured by electrographic and behavioral seizures.
Interestingly, F4/80High cells from CLP-operated mice exhibited a higher phagocytic activity than those from sham-operated mice.
Young euglycemic Dpp4 mice exhibited a cardioprotective response after TAC surgery or doxorubicin administration, with reduced fibrosis; however, cardiac mRNA analysis revealed increased expression of inflammation-related transcripts.
Furthermore, when compared with IL-7R-deficient mice, anti-gamma c chimeric and gamma c-deficient mice exhibited a distinct phenotypic pattern of pro-T cell development.
KO mice exhibited a significant increase of paracellular permeability.
