Sentence examples for mice excluding an from inspiring English sources

The numbers of total thymocytes and the numbers of each thymocyte subset in Lck-cre mice were similar to the numbers observed in DGf/f mice, excluding an adverse effect of the cre transgene expression on T cell development in our system (Fig. S3).

And the treatment with control siRNA did not alter cerebral NLRP1 mRNA and protein levels compared with No siRNA-treated APP/PS1 mice, excluding an effect of pump-mediated infusion on NLRP1 expression levels.

However, the growth of Salmonella was similar in the plasma of LDLR−/−, ApoE−/− and control mice, excluding a direct antimicrobial effect of LDL.

Generation of TLR2/C3 double-deficient mice gave similar responses as TLR2-/ mice, excluding a synergistic effect of double deficiency and confirming no role for the alternative pathway component of the complement cascade (Fig. 4a).

Finally, sham-operated and thymectomized mice subjected to antibody treatment and subsequent immunization appeared equally protected from CIA, compared with nondepleted mice, excluding a role for recent B22a1+CD4+ thymic emigrants in the later-occurring arthritis (data not shown).

The expression of those genes was very low in both iWAT and eWAT and not increased in C/EBPβΔuORF/BL6 mice excluding a critical involvement of uncoupling or browning of WAT as a cause of the higher energy expenditure.

Neither epidermal nor dermal LC numbers were significantly changed between pDC-sufficient and pDC-depleted DKO* mice, excluding a counterregulation of LC numbers through pDCs (Supplementary Fig S3O and P).

We did not see changes in dimensions of the OC of the Cdc42 loxP/loxP Fgfr3-iCre-ER T2 mice, excloxP/loxP Fgfr3-iCre-ERor Cdc42 in the reguloxP/loxP Fgfr3-iCre-ERnT2 in the cochlea.

Our data revealed a somewhat similar tibial length among young and aging C57 or ob/ob mice, excluding the possible contribution of disparate growth and development in these mice.

Fasting glucose levels and systolic blood pressure were similar between WT and APP/PS1 mice, excluding the potential contribution of diabetes and hypertension.

Identifying the DRP-1β isoform in the brain of embryonic and young mice excluding the adult phase may have functional implications for our future understanding of why such domain modularity evolved in this family of death –inducing kinases.