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However, oxygen consumption and locomotor activity were not significantly different between CXCL14+/− and CXCL14−/− mice either at night or during the daytime (Figure 3C, E).
Unlike phase delay, phase advance elicited by a light pulse at late subjective night (5 min, CT21) was not altered in Adcyap1−/− mice, either at 20 or 100lx (Fig. 1C).
Body weight, fasting blood glucose, and glucose area under the curve after ip GTT were not significantly different in WT, 5αR1−/−, and 5αR2−/− mice either at baseline or at 6 or 12 months of NC feeding (Table 1).
Fibers from WT mice treated with l-NMMA showed no differences in the rate of DHE oxidation compared with fibers from WT mice either at rest, or following contractile activity.
Using a preventive treatment paradigm, they observed that, while DMF administration in wild-type mice strongly downregulated disease expression as expected, it had no effect on EAE induced in Hcar2−/− mice, either at the clinical or neuropathological level.
No significant difference in the % CD8 T cells in secondary lymphoid organs or peripheral blood was observed in Idd9 congenic mice either at 6 weeks of age, or in older mice (data not shown).
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In order to limit the number of blood draws per mouse, groups were split in two and blood was taken only once a day for each mouse (either at 2 hours or 12 hours post DPPIV inhibitor administration).
Five days after tumor inoculation, mice received either AT-101 (50 mg/kg, intraperitoneally, five times per week) or an equal volume of vehicle.
EL4 lymphoma-bearing mice either were cured at higher doses of radioimmunotherapy alone or lower doses of radioimmunotherapy in synergy with chemotherapy.
Two experiments were performed, with mice either being killed at 3 months of age or aged until tumour development.
The same dose of 750 cGy was given to CBA mice either in a single dose at a low dose rate (4 cGy min-1) (STBI) or in a fractionated regimen (six fractions of 125 cGy three times a day) at a higher dose rate (25 cGy min-1) (FTBI).
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