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In addition, male PI4KIIα TG mice displayed relatively worse glucose tolerance after STZ/HFD treatment, with more rapid progression of diabetes compared to WT mice; the phenotype of PI4KIIα TG mice after 2 weeks was similar to that of WT mice after 3 weeks (Fig. 6D).

However, this was not found to be the case, as the ICR-GSK-3α Alb Cre + mice displayed relatively normal insulin sensitivity.

Third, despite this broad expression, both Lrig1 and Lrig3 mutant mice display relatively mild phenotypes.

Accordingly, ACKR2-deficient mice display relatively inefficient antigen presentation from inflamed sites.

Palatal suture scoring and comparative 3D shape analysis from high resolution μCT images of 120 newborn mouse skulls showed that Fgfr2 +/S252W mice display relatively more severe palate dysmorphologies, with contracted and more separated palatal shelves, a greater tendency to fuse the maxillary-palatine sutures and aberrant development of the inter-premaxillary suture.

Interestingly, a "mild disease-like" pattern appeared in a smaller number of mice (10/46), which displayed relatively low bacteremia levels of less than 106 CFU/ml (Figure 2B).

In the CT mice, cells from all three groups displayed relatively high lap-by-lap co-variation during DisjRun1 exploration.

Chronic phase ex vivo GIT bioluminescence was typically highly focal, but displayed relatively low variation in intensity between mice, indicating a numerically stable but spatially dynamic parasite load in this organ.

These methods generally displayed relatively high detection limits.

The two-step assay displayed relatively higher S/Co ratios.

All displayed relatively recent activity, <40 Myr (table 3).